New Approaches to Aortic Diseases from Valve to Abdominal Bifurcation.

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Bibliographic Details
Main Author: Tintoiu, Ion C.
Format: Electronic eBook
Language:English
Published: Saint Louis, UNITED STATES : Academic Press, 2017.
Subjects:
Online Access: Full text (Emmanuel users only)
Table of Contents:
  • Front Cover; New Approaches to Aortic Diseases from Valve to Abdominal Bifurcation; Dedication; New Approaches to Aortic Diseases from Valve to Abdominal Bifurcation; Copyright; Contents; List of Contributors; Preface; 1
  • Overview; 1
  • Aging Aortaâ#x80;#x94;Cellular Mechanisms; AGING AND STIFFNESS AORTA; INTIMAâ#x80;#x93;ENDOTHELIAL SENESCENCE; INTIMA AND MEDIAâ#x80;#x94;APOPTOSIS; INTIMAâ#x80;#x93;EXTRACELLULAR MATRIX AND MMPS ROLES; MEDIAâ#x80;#x93;VSMCS; MEDIAâ#x80;#x93;ELASTIN/COLLAGEN RATIO; MEDIA INFLAMMATION; TELOMERE LENGTHâ#x80;#x94;A BIOMARKER OF AORTA AGING; ATHEROGENESIS; CONCLUSIONS; REFERENCES; FURTHER READING.
  • 2
  • Endothelial Dysfunction in Aortic AneurysmOVERVIEW; OXIDATIVE STRESS; GRAFT ENDOTHELIALIZATION; CONCLUSIONS; REFERENCES; 3
  • Histology of Aortic Disease and Progression of Aortic Dissection From Acute to Chronic; INTRODUCTION; NORMAL HISTOLOGY OF THE AORTA; HISTOLOGY OF AORTIC ANEURYSM AND DISSECTION; HISTOLOGY OF THE TRANSITION FROM ACUTE TO CHRONIC DISSECTION; MACROSCOPIC CHANGES OF THE TRANSITION FROM ACUTE TO CHRONIC DISSECTION; Stanford Type B Dissections; Expansion of the Dissected Aorta; Progression of the Dissection; False Lumen Patency; Intimal Flap Architecture and Its Dynamics.
  • Stanford Type A DissectionFalse Lumen Patency; REFERENCES; 4
  • The Pathogenesis of Aortic Valvular Disease; PATHOGENESIS OF AORTIC STENOSIS; The Inflammatory Process; The Lipoprotein Anomalies; The Activation of Reninâ#x80;#x93;Angiotensin System; Calcification in Aortic Stenosis; AORTIC REGURGITATION; REFERENCES; 5
  • Aortic Root Pathologies; INTRODUCTION; FUNCTIONAL ANATOMY; HISTOPATHOLOGIC CHANGES; AORTIC ROOT PATHOLOGIES; Aging; Aneurysm; Poststenotic Dilatation; Sinus of Valsalva Aneurysm With/Without Rupture; Pseudoaneurysm; Dilatation Associated With Congenital Heart Diseases.
  • Aortic Root AbscessAorto-Left Ventricular Tunnel; Porcelain Aorta; Supravalvular Stenosis; Aortic Dissection; REFERENCES; 6
  • Cellular Mechanisms of Ascending Aortic Aneurysms; STRUCTURE, SIZE, AND LOCATION OF AORTA; DIAGNOSIS AND RISK FACTORS OF ASCENDING AORTIC ANEURYSM; CHARACTERIZATION OF ASCENDING AORTIC ANEURYSMS; CELLULAR MECHANISMS OF ASCENDING AORTIC ANEURYSM; Endothelial Cells; Smooth Muscle Cells; Fibroblasts; Immune Cells; Elastin; Collagen; Microfibrils and Fibrillin; Fibulin; ADDITIONAL FACTORS INVOLVED IN ASCENDING AORTIC ANEURYSM.
  • POTENTIAL DRUG TREATMENTS FOR ASCENDING AORTIC ANEURYSMSUMMARY; ACKNOWLEDGMENTS; REFERENCES; 7
  • Mathematical Modeling of Aortic Aneurysm Progression; INTRODUCTION; MATHEMATICAL MODELING AND SYSTEMS ANALYSIS OF BIOLOGICAL PHENOMENA; PETRI NETâ#x80;#x93;BASED MODEL OF AORTIC ANEURYSM PROGRESSION; ACKNOWLEDGMENTS; REFERENCES; 8
  • Genetic Basis of Aortic Disease; SYNDROMIC THORACIC AORTIC ANEURYSMS AND DISSECTIONS; Marfan Syndrome; Genetic Basis and Molecular Mechanisms; Loeysâ#x80;#x93;Dietz Syndrome; Genetic Basis and Molecular Mechanisms; Ehlersâ#x80;#x93;Danlos Syndromes; Genetic Basis and Molecular Mechanisms.